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Previously, we have shown that C2C12 mouse myoblasts genetically modified to express human LMNA (hLMNA) gene bearing R482L mutation exhibited alterations in coordination of cell cycle dynamics and muscle differentiation, mitochondrial uncoupling, and a decrease in glycolytic activity in skeletal muscle myofiber 18,19,20

Nazir, 2024)

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The 5-year LRR is 2.3%, with an OS of 97.2%

Although NADPH was not directly measured, this inferred increase is consistent with the observed downregulation of inflammatory gene expression in the liver and may reflect improved redox buffering capacity